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Emerging data in clinical and preclinical studies suggests the adverse and efficacious effects of these pharmacotherapies can be dissociated, and that GIPR agonism improves the GI AE profile of GLP-1R and dual incretin receptor agonists

10,11 The etiology, physiology and microbiology of ARDS are highly heterogeneous, 10 so personalized medicine approaches for patients with different phenotypes might be a goal of future treatment